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 Biochemistry Mnemonics Only!  

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Tay-Hex instead of Tay-Sachs (no hexosaminidase)
Glucher instead if Gaucher (no glucocerebrosidase)


Ehlers-Danlys instead of Ehlers-Danlos ! Can u tell me why?


Danlys, maybe because it involves collagen and you have lysine in collagen??


Yes ! Lys comes from lysine . There must be some problems with lysine...
ED syndrome is due to a defect of the gene for Lysine Hydrohylase, and the lysine is not hydrohylated.Result: impaired collagen functions) (HY- Histology -ch.3 Connective tissue)


Must remember adenine binds with thymine and cytosine with guanine
from Biology 101.(except uracil for rna)
For the # of H bonds:

*2 Thighs and 3 Good Chickens

AT-2 hydrogen bonds
GC-3 hydrogen bonds


Proline and Lysine residues of collagen are hydroxylated by proline hydroxylase and lysine hydroxylase right after synthesis and this modification is for proper connective tissue function of Collagen. These 2 enzymes require 4 cofactors; Fe++, O2, Vit C & alpha-ketoglutarate. So I'd say; "Keep Our Collagen Fine" to memorize these molecules :wink:
Just to remind; Lysine Hydroxylase gene is mutated in Ehlers Danlos, also know as "Ehlers Danlys" smiling face :arrow: hyperelasticity and fragility of the skin, hypermobility of the joints, and fragility of the cutaneous blood vessels, etc.


ToMYs GirlFrind is AHa!!

everywhere it is repeat of C__G , where __ stands for:

T(CTG) : in Myotonia dystrophica

G(CGG): in FRagile x syndrome

A(CAG) : Hungtons chorea and other disorder

ToMYs GirlFrind is AHa!!

ny new improvements r always welcome.


I need to memorize the structures (not just the hydrophobicity, esstential, etc, I need to be able to draw them).

Anyone know any good tricks to memorizing the structures for all 20 amino acids?



How do you link freckles, Thymine-Thymine dimers, and UV light?

Think of Peppermint PaT-Ty (freckles) taking a UV light bath. 8)


Essential amino acids; The Life ARGentina’s Maradona LEd IS TRYP, HIStorical, PHYlosphical, VALorous


Do Enter After the Bell

Cyclin D/E - G1 :arrow: S
Cyclin A - S :arrow: G2
Cyclin B - G2 :arrow: M


For Eukaryotic Cells- DNA Replication

U all probably already thought of this but just wanted to put it down for my satisfaction: THANX!!

LeaDing strand- dna polymerase Delta

LAgging strand- dna polymerase Alpha


Let me repeat the last part of that one again:

LAgging Strand- dna polymerase Alpha


I always forget competitive and non-competitive inhibition on linewiever burk i made this.

in competitive inhibtion there is always competetion between two lines so they cross on graph.So solute concentration chages but velocity remains same.vice versa for non competitive.


km always higher than vmax like either km inc and vmax same or km same vmax dec

kmpitative inhibitor ==km
increase ,vmax same

non kmpitative inhibitor==== km not increased, vmax decreased

Edited by drkaur on Sep 18, 2005 - 9:35 AM


drstepbystep wrote:

How do you link freckles, Thymine-Thymine dimers, and UV light?

Think of Peppermint PaT-Ty (freckles) taking a UV light bath. 8)

what is this for?in regards to what?ciould you clarify?



n=number of carbons , N=n/2

ATP=(12xN) + (5x(N-1)) -2


Learning the Cofactors for Enzymes is Painful so you hit them with a BAP!
BAP! Tender Loving Care for Noone!

(BAP needs TLCFN)

Branched Chain Ketoacid DH
Alpha ketoglutarage DH
(by the way, all are DH enzymes; ALL DH enzymes produce NADH)

they need:

TTP from Thiamine (B1)
Lipoic Acid (Synthesized; not from a vitamin)
CoA (from Pantothenate)
FAD(H2) from Riboflavin (B2)
NAD(H) from Niacin (B3)

To remember what TLCFN comes from its: BSPRN (bull sh.. prn)


Another one:

Carboxylases need ABC:
ATP, Biotin, CO2

All Dehydrogenases: make NADH

All Kinases: Phosphorylate (KINdly put a phosphate on)

All Phosphatases: DEphosphorylate (PhosphaTASE, TAKES off Phosphate)

Glucagon puts Phosphate ON (phosphorylates); and INversely INsulin does the opposite (dephosphorylate)

This is a good way to remember what enzymes of which pathways are active/inactive in phosphorylated/dephosphorylated form

eg. Insulin activates Glycolysis, and Glycogen synthesis, so the enzymes that it activates in those pathways will be active in DEphosphorylated form.

and: Glucagon activates Glycogenolysis and Gluconeogenesis, so the enzymes that it activates in those pathways will be active in Phosphorylated form.


A little tragic no one has taken up the Peppermint patty question.

My guess is it refers to Xeroderma pigmentosa, an autosomal recessive disorder of DNA repair. Humans typically utilize DNA excision repair, etc. to repair Thymidine dimers caused by exposure to UV light (UVB). There does exist another utilization to specifically fix these dimers called DNA photolyase, however if ever on a question this answer MUST be wrong since humans do not produce photolyase. Mutations in DNA excision repair mechanisms on the XP genes can cause build-up of thymidine dimers, which causes DNA damage, which of course leads to mutations and problems of expression. The end result is these patients have freckles, photosensitivity, and a propensity to have "paper-like skin". They also develop Basal and Squamous carcinomas and Actinic keratoses.

Mneumonic: Pepermint Patty, in a UV bath, with Windows XP.


PPG= Pyruvate, phosphofructo,gluco kinase= 3 irreversibile reaction in glycolysis.
Preaty Please Go!

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