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NNL Awaitin The Unexpected Topics: 779 Posts: 6613	Apr 18, 2007 - 6:27 PM       #1 We All Love the Author But Mistakes can Happen by Anyone So here is a List of His Own Updated Errata. Page 41 Under 1. Acute inflammation (see Fig. 2-13A) Under 2. Chronic inflammation (see Figure 2-13B) Edited by new_n_lost on Apr 18, 2007 - 7:14 PM
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NNL Awaitin The Unexpected Topics: 779 Posts: 6613	Apr 18, 2007 - 6:28 PM       #2 Page 106 Margin note Triple marker for Down syndrome: decreased AFP, increased hCG, decreased urine estriol Edited by new_n_lost on Apr 18, 2007 - 7:14 PM
NNL Awaitin The Unexpected Topics: 779 Posts: 6613	Apr 18, 2007 - 6:31 PM       #3 Page 128 Under D. Vitamin K, 3. Function a. g-Carboxylates glutamate residues in vitamin K-dependent procoagulants and anticoagulants (protein C and S) (1) Procoagulants include factors II (prothrombin), VII, IX, and X (delete protein C and S, which are anticoagulants) Edited by new_n_lost on Apr 18, 2007 - 7:14 PM
NNL Awaitin The Unexpected Topics: 779 Posts: 6613	Apr 18, 2007 - 6:35 PM       #4 Page 129 Table 7-3 Thiamine (vitamin B1) Korsakoff’s psychosis: antegrade and retrograde Edited by new_n_lost on Apr 18, 2007 - 7:15 PM
NNL Awaitin The Unexpected Topics: 779 Posts: 6613	Apr 18, 2007 - 6:37 PM       #5 NEOPLASIA Page 138 D. Growth rate 3. Thirty doubling… · Equivalent to 109….. Page 142 Table 8-1 Autosomal dominant cancer syndromes Retinoblastoma (two-hit theory) Page 145 5. DNA repair genes (see Table 8-1 and Table 8-4) Edited by new_n_lost on Apr 18, 2007 - 7:16 PM
NNL Awaitin The Unexpected Topics: 779 Posts: 6613	Apr 18, 2007 - 6:45 PM       #6 Page 162 Table 9-1 Under Sturge-Weber syndrome Delete (AD) Actually, it is now considered somatic mosacism Edited by new_n_lost on Apr 18, 2007 - 7:16 PM
NNL Awaitin The Unexpected Topics: 779 Posts: 6613	Apr 18, 2007 - 6:47 PM       #7 Heart Disorders Page 173 Under Prinzmetal’s angina Increase in endothelin Page 182 Tetralogy of Fallot (7) Tet spells (a) Sudden increase in hypoxemia and cyanosis Edited by new_n_lost on Apr 18, 2007 - 7:17 PM
NNL Awaitin The Unexpected Topics: 779 Posts: 6613	Apr 18, 2007 - 6:49 PM       #8 RBC Page 203 Under C. 3. (c) Microcytic and normocytic cells with increased central area of pallor (Fig. 11-6) Edited by new_n_lost on Apr 18, 2007 - 7:17 PM
NNL Awaitin The Unexpected Topics: 779 Posts: 6613	Apr 18, 2007 - 6:55 PM       #9 RESPI Page 300 Table 16-3 Put Chlamydia, Rickettsia, Mycoplasma under Bacteria not Viruses heading Page 323 Table 16-5 Under Bronchial hamartoma Put- Peripheral (90%), Central (10%) Edited by new_n_lost on Apr 18, 2007 - 7:18 PM
NNL Awaitin The Unexpected Topics: 779 Posts: 6613	Apr 18, 2007 - 7:02 PM       #10 GI Page 352 Table 17-7 Under Hernia Direct Medial border of triangle is rectus abdominis muscle, lateral border is inferior epigastric artery, inferior border is inguinal ligament Page 358 c. Peutz-Jeghers polyposis (1) Autosomal dominant (2) Polyps predominate in the small bowel; less common in stomach and colon MN: Peutz-Jeghers polyposis: predominance of small intestine polyps In Anderson’s textbook of Pathology and Sabiston’s Surgery, Peutz-Jeghers polyps are listed as hamartomas, because they are a non-neoplastic overgrowth of normal tissue. In all the major texts, the small intestine is listed as the most common site for the polyps. They are less common in stomach and colon. Key point is that it is the only hereditary polyposis syndrome with polyps more common in the small intestine than the colon. Juvenile polyps are listed as hamartomatous polyps in Anderson’s Pathology and Morson Gastrointestinal Pathology. They are sometimes called retention polyps. Hyperplastic polyps are non-neoplastic polyps that some purists consider hamartomas and others a type of metaplasia, with mucosa resembling small bowel. The key point is that they are non-neoplastic and do not transform into cancer. Edited by new_n_lost on Apr 18, 2007 - 7:18 PM
NNL Awaitin The Unexpected Topics: 779 Posts: 6613	Apr 18, 2007 - 7:11 PM       #11 Hepato - Biliary Page 371 Under e. Chronic HBV, (1) …converts to anti-HBc-IgG Page 375 Margin Note Fulminant hepatic failure: ¯ transaminases, ­ PT and ammonia Pages 377 and 378 Under A. Alcohol related disorders 1. Should say (Chapter 6) 3. Should say (Chapter 6) 4. Should say (Chapter 6) Edited by new_n_lost on Apr 18, 2007 - 7:21 PM
NNL Awaitin The Unexpected Topics: 779 Posts: 6613	Apr 18, 2007 - 7:21 PM       #12 Renal Page 396 Table 19-1 See the Attached Doc for the Table I moved the causes of glomerular proteinuria to line-up with nephritic and nephrotic. The original table was not intended to match up the cause of glomerular proteinuria with an example of a nephritic syndrome and nephrotic syndrome. Because this has caused confusion, I changed diffuse membranous glomerulopathy, the most common cause of nephrotic syndrome in adults, to post-streptococcal glomerulonephritis and lined this up with nephritic syndrome and minimal change disease, the most common cause of nephrotic syndrome in children, with nephrotic syndrome. In the original table, it looked like I was implying that minimal change disease was an example of glomerular injury in the nephritic syndrome, which is of course, incorrect. Page 395 B. Serum creatinine 2. Creatinine is filtered in the kidneys and not reabsorbed or secreted This sentence is a generality and is true at the normal concentration of creatinine. It was not part of a discussion of renal failure or of variations of creatinine in emaciated individuals versus muscular people or the effect of drugs on the measurement of serum creatinine. It is true that at high serum levels of creatinine, some is secreted into the urine, so it overestimates the creatinine clearance in renal failure; however, many other tests help corroborate the severity of the renal failure like serum calcium, phosphorus, BUN, and electrolytes showing hyperkalemia and metabolic acidosis. However, irrespective of this, the creatinine clearance is a more practical and cost effective test even though it is not a perfect clearance substance like inulin. Page 409 Table 19-7 Change IgA glomerulonephritis to IgA glomerulopathy Page 412 Table 19-8 Type I MPGN Subendothelial ICs Attached Files: Table 19-1.doc (25 KB, 51 downloads) Edited by new_n_lost on Apr 18, 2007 - 7:45 PM
NNL Awaitin The Unexpected Topics: 779 Posts: 6613	Apr 18, 2007 - 7:46 PM       #13 Page 443 3. Prolactin a. Prolactin enhances testosterone function and spermatogenesis. A number of articles discuss the important role that prolactin normally has in promoting the function of the testis and accessory structures. Prolactin receptors are present in the Leydig cells, differentiating germ cells in the seminiferous tubules, and all of the accessory structures. Specifically, in the Leydig cells, prolactin is important in inducing and maintaining LH receptors; which enhances testosterone function. It also directly has an effect on enhancing spermatogenesis. When prolactin is increased, it shuts off GnRH; which, in turn, decreases testosterone production (LH effect) and spermatogenesis (FSH) effect.
NNL Awaitin The Unexpected Topics: 779 Posts: 6613	Apr 18, 2007 - 7:49 PM       #14 Page 446 (not an errata; just a better term) IX. Erectile dysfunction Drop the definition underneath since it is self-explanatory Page 479 Under 3. Traumatic fat necrosis c. Painless, indurated mass add: · Painful in acute stage Page 504 Table 22-3 Pseudohypoparathyroidism: autosomal dominant Page 505 Clarification Hypertension in hypercalcemia: Hypertension is seen with increased frequency in patients with hypercalcemia (e.g., primary hyperparathyroidism). It may be caused by renal insufficiency and/or calcium-mediated vasoconstriction of arterioles. Page 516 (not an errata; just an additional designation) C. Syndrome X (metabolic syndrome) Page 532 Under Septic arthritis, change C5-C9 to C6-C9. Page 537 Table 23-3 Compartment syndrome Volkmann’s ischemic contracture: supracondylar fracture of humerus…. Page 543 Under A. Staphylococcus aureus skin infections Add d. Impetigo Page 544 Under C. Impetigo 1. Most often caused by Staphylococcus aureus · Streptococcus pyogenes second most common cause 3. Presence of bullae commonly occurs with Staphylococcus aureus Page 551-552 2. Pemphigus vulgaris (page 551) c. Intraepithelial vesicles are located above the basal layer (suprabasal). (3) Positive Nikolsky sign · Outer epidermis separates from basal layer with minimal pressure 3. Bullous pemphigoid (page 552) b. Vesicles are subepidermal. (3) Negative Nikolsky sign The oral mucosa is involved in one-third of cases, which is not rare as one medical student suggested on an Internet site. This was a really bad mistake in editing, because I had in correct in the notes I used to teach students with before the book came out. Page 569 Under 4. Sturge-Weber syndrome a. Somatic mosaicism or sporadic It is not autosomal dominant Page 578, Table 25-2 Under Rabies virus: Most often transmitted by raccoon bite (40%) Other vectors are dog, skunk, bat, coyote Page 579 Under Table 25-4: Treponema pallidum Argyll-Robertson pupil (pupils accommodate but do not react) Page 583 2. Role of beta-amyloid (Abeta) protein b. Defects in degradation of APP by secretases cause an increase in Abeta (1) Alpha-secretases cleave APP into fragments that cannot produce Abeta (2) Beta and gamma-secretases cleave APP into fragments that are converted to Abeta. Beta secretases first must cleave (“clip”) the APP and then subsequent cleavage by gamma secretases produces Abeta, which forms amyloid that is neurotoxic. Page 585 Figure 25-17 Coronal section (B) shows (leave out dilated lateral ventricle and) atrophy of the caudate, putamen, and globus pallidus when compared with a normal coronal section (A). A different picture was placed in there during publication, but the original discussion was not changed. The above is a correct discussion of the new picture. Page 586 Under F. Werdnig-Hoffman disease · Lower motor neuron disease that occurs in children. QUESTION 10 (Page 652 has question, page 657 has discussion) A 9-month-old girl has an infection on her face that began as erythematous macules. She later develops pustules that rupture and cause honey-colored crusted lesions. The girl's 5-year-old brother develops similar lesions. Which of the following is the causal agent? A. Staphylococcus aureus B. Herpes simplex virus type 1 C. Malassezia furfur D. Propionibacterium acnes E. Trichophyton rubrum A (Staphylococcus aureus) is correct. The child has impetigo, which causes honey-colored crusted lesions that cover shallow ulcerations of the skin. Staphylococcus aureus is the most common cause of this superficial skin lesion. Streptococcus pyogenes is the second most common cause of impetigo. Impetigo is highly contagious, which explains why the child's brother develops similar lesions. B (herpes simplex virus type 1) is incorrect. Herpes simplex virus type 1 produces vesicles and pustules on the vermilion border of the lip. It does not produce honey-colored crusted lesions on the face. C (Malassezia furfur) is incorrect. M. furfur, a superficial dermatophyte, causes tinea versicolor and seborrheic dermatitis (dandruff). It does not produce honey-colored crusted lesions on the face. D (Propionibacterium acnes) is incorrect. P. acnes is an anaerobe involved in producing the inflammatory reaction associated with acne vulgaris. It does not produce honey-colored crusted lesions on the face. E (Trichophyton rubrum) is incorrect. T. rubrum, a superficial dermatophyte, causes tinea corporis (body), tinea cruris (groin), and tinea pedis (foot). It does not produce honey-colored crusted lesions on the face. Edited by new_n_lost on Apr 18, 2007 - 7:56 PM
ShweHope99.9998 Forum Fanatic Topics: 12 Posts: 2763	May 19, 2007 - 9:13 PM       #15 Thanks
NNL Awaitin The Unexpected Topics: 779 Posts: 6613	May 24, 2007 - 5:48 PM       #16 Plz Chk the Table on PG 396 Table 19-1 NEPHROTIC SYN HAS SELECTIVE PROTIENURIA & LOSS OF ALBUMIN ONLY WHEREAS IN NEPHRITIC SYN WE HAVE NON SELECTIVE LOSS OF PROTEINS ALBUMIN N GLOBULINS, TOO Attached Files: Table 19-1.doc (25 KB, 36 downloads) Edited by new_n_lost on May 24, 2007 - 5:55 PM
mylife_step1 Forum Newbie Topics: 3 Posts: 10	Jun 05, 2007 - 7:30 AM       #17 thanks doc. great job
hedieh Forum Guru Topics: 47 Posts: 432	Aug 17, 2007 - 8:17 AM       #18 Thanks 1000 times, New_n_lost, great job. I found another mistake and posted it in a separate topic to be noticed.
yoga Forum Guru Topics: 73 Posts: 637	Aug 18, 2007 - 4:31 AM       #19 thanx nnl ki have suggestion ,,why dont u email the author & give ur correction? usually they $$$$$ u will recieve $$$ & acknowledgement in next edition
edie Forum Guru Topics: 26 Posts: 1198	Aug 18, 2007 - 5:44 AM       #20 Some students contacted himabout this when the book came out last fall. Dr Goljan has a file for errata for download here: www.healthsciences.okstate....dical/pathology/goljan.cfm

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