paheli
It'sAllAboutGoodKarma
Topics: 197
Posts: 2,720
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Sorry, I don't understand your qn.
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| hero
Forum Guru
Topics: 42
Posts: 557
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I think it is about dopamine - serotonin
negative symptoms respond to atypical antypsichotics better, which are blocking serotonin receptors and adrenoreceptors
(more negative symptoms associated with poorer prognosis)
positive symptoms - respond to typical antipsichotics - they blocking dopamine receptors
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| Vietnamese
Forum Elite
Topics: 12
Posts: 285
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Long time ago, I heard negative symptoms result from temporal atrophy and positive symtoms result from hippocampal atrophy. These concepts are very important to control symptoms.
That's right?
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| hero
Forum Guru
Topics: 42
Posts: 557
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don't know about that. KLN sayes about ventricular enlargement and something about hyperactivity in basal ganglias (where dopamine concentrates) and hypoactivity in frontal lobes.
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| Anand007
Forum Senior
Topics: 5
Posts: 118
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UW says..enlarged ventricles, prominent sulci.. decreased cerebral, hippocampal and temporal masses
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| cingulate
Forum Newbie
Topics: 5
Posts: 21
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the most important thing is that
increase mesolimbic activity--------positive symptoms
decrease mesocortical activity-------negative symptoms
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