Summer.Thunder
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45, woman, acute diverticulitis, develops severe hypotension, tachypnea, tachycardia, cyanosis, hypoxemia, unresponsive to Oxygen therapy, x-ray shows diffuse alveolar infiltrate, blood grows e coli,
major factor is: relase oxygen free radicals from sequestered neutrophils
can someone explain this to me? thanks
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| sidiyaa
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plz can give us all the choice
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| angel23
Forum Guru
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this is a case of ARDS
pathogenesis of ARDS is destruction by neutrophils primarily.....so this choice
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| angel23
Forum Guru
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refer goljan rapid review
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| antidepressant
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this discussion was posted in micro, it should rather be here, explained better, its in RR too, but forgot, good question.
http://www.prep4usmle.com/forum/thread/36721/
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| antidepressant
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From Papi's lecture:
Adult Respiratory Distress Syndrome (ARDS)
In terms of ARDS, essentially it is the same as RDS(in child) in pathophys, but is NEUTROPHIL related injury. In RDS you’re not making surfactant b/c you are too premature or have too much insulin and just have collapsed alveoli. BUT in ARDS its b/c you have too much inflammation; there is no inflammation in RDS.
MCC ARDS = septic shock (MCC septic shock = E coli, due to sepsis from an indwelling catheter; MCC DIC = septic shock). Example: In the ICU – if a pt come in with dyspnea and its within 24 hrs of having septic shock, pt has ARDS. If pt is in septic shock within 48 hrs and is bleeding from every orifice, he has DIC. So, first day = septic shock, second day = ARDS, third day = DIC.
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Step 1 can not defeat me!!!!!!!!
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