new_n_lost wrote:
c) Fissuring of the plaque in left coronary artery with superimposed mural (partial) thrombosis

Pathophysiologic Events Culminating in the Clinical Syndrome of Unstable Angina

Numerous physiologic triggers probably initiate the rupture of a vulnerable plaque. Rupture leads to the activation, adhesion, and aggregation of platelets and the activation of the clotting cascade, resulting in the formation of an occlusive thrombus. If this process leads to complete occlusion of the artery, then acute myocardial infarction with ST-segment elevation occurs. Alternatively, if the process leads to severe stenosis but the artery nonetheless remains patent, then unstable angina occurs.