kity
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major site of airway resistance???
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| kity
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why chronic bronchitis---blue bloater??
& emphysema ---------pink puffers??
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| hope4dabest
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kity wrote:
why chronic bronchitis---blue bloater??
& emphysema ---------pink puffers??
due to vebtilation perfusion defects
chr bronchitis---hypoxia, polycythaemia and CO2 retention
emphysema---gud bld flow through less oxygeneated lung
max resistence----arterioles
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| hope4dabest
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which virus causes Reyes syndrome or GBS??
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| anuba
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H influenza type b or chicken pox virus
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| kity
Certified.............
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[quote=hope4dabest]
due to vebtilation perfusion defects
chr bronchitis---hypoxia, polycythaemia and CO2 retention
emphysema---gud bld flow through less oxygeneated lung
max resistence----arterioles
[/quot
I am still confused why emphysema are pink.. if good blood flow is the reason... we all shud be pink... 
& I was talking abt max airway resistance...??
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| kity
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hope4dabest wrote:
which virus causes Reyes syndrome or GBS??
reye syndrome -chicken pox.. I guess using salicylates in any viral did can cause it
GBS-CMV, EBV, herpes, influenza vaccine
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| hope4dabest
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emphysema--hyperventilate to maintain adequate blood oxygen levels... due to Hyperventilation mild emphysema patients do not appear cyanotic as chronic bronchitis patients do, hence they are "pink puffers" (able to maintain almost normal blood gases through hyperventilation)
didnt read the q properly......so careless of me
max airway resistence----is it terminal bronchioles???
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| amritt786
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kity wrote: reye syndrome -chicken pox.. I guess using salicylates in any viral did can cause it GBS-CMV, EBV, herpes, influenza vaccine
which bacteria causes GBS?
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| amritt786
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aneurysm of internal carotid artery cause which kind of visual defect?
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| amritt786
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lets revise visual defect
1 visual defect due occulusion of post cerebral artery?
2 visual sefect in optic neuritis? and what r symtoms of optic neuritis?
3 v defect in lesion of myeres loop and lesion optic radiation to cuneus?
4 what is cortical blindness?
5 what is argyl robertson pupil?`
6 optic tract lesion due to craniphrangioma and pituatary adenoma?
so plz post as many as question from single topic so that we can cover almost all parts of that topic ...... espacially points which r volatile and need cramming thanks
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| kity
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amritt786 wrote:
which bacteria causes GBS?
campylobacter..
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| kity
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amritt786 wrote:
aneurysm of internal carotid artery cause which kind of visual defect?
nasal hemianopia
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| medocuk
IM-RES
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1. Homonymous hemianopia with macular sparing.
2. Anopsia, generally due to Multiple sclerosis.
3. Meyer's loop: Contralateral Superior quadrantanopia,
Optic radiations: COntralateral Inferior quadrantanopia
4. Blindness with pupillary reflexes intact
5. Loss of pupillary reflex with accomodation still intact. Seen in neurosyphillis, MS, pineal glad tumors.
6. Affecting the optic chiasma: Bitemporal hemianopia.
Question regarding ICA aneurysm: Occulomotor nerve palsy: Ptosis,mydriasis, loss of accomodation, loss of motor limb of pupillary reflex and most importantly loss of adduction.
My question: What apart from Increased AFP does one detect in neural tube defects.
What is Internuclear ophtalmoplegia.
amritt786 wrote:
lets revise visual defect
1 visual defect due occulusion of post cerebral artery?
2 visual sefect in optic neuritis? and what r symtoms of optic neuritis?
3 v defect in lesion of myeres loop and lesion optic radiation to cuneus?
4 what is cortical blindness?
5 what is argyl robertson pupil?`
6 optic tract lesion due to craniphrangioma and pituatary adenoma?
so plz post as many as question from single topic so that we can cover almost all parts of that topic ...... espacially points which r volatile and need cramming thanks
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| kity
Certified.............
Topics: 31
Posts: 1,067
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amritt786 wrote:
lets revise visual defect
1 visual defect due occulusion of post cerebral artery?
2 visual sefect in optic neuritis? and what r symtoms of optic neuritis?
3 v defect in lesion of myeres loop and lesion optic radiation to cuneus?
4 what is cortical blindness?
5 what is argyl robertson pupil?`
6 optic tract lesion due to craniphrangioma and pituatary adenoma?
so plz post as many as question from single topic so that we can cover almost all parts of that topic ...... espacially points which r volatile and need cramming thanks
1.hemianopia of opp side with macular sparing
2. blindness in the same eye.. will cause pain too
3 supireor quadrantopia
4 cerebral cause of blindness.. pt unaware of the defect..
5 pupil accomodates but doesnt constricts.. seen in syphilis
6 bitemporal hemianopia
how do you diff cortical blindness from other causes
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| kity
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kity wrote:
major site of airway resistance???
medium bronchi
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| amritt786
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great job medocuk and kity
about question inc AFP in NTD i have point in my subconcious mind but can not come out....
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| mytime
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kity wrote:
major site of airway resistance???
Resp. bronchiles.
Do u guys ever sleep???????????????
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| hope4dabest
Forum Elite
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i do..but see kity wants to have Pain so that he/she can Gain
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| hope4dabest
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kity wrote:
medium bronchi
i knpw it sounds dumb....but whats medium bronchi????
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| kity
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mytime wrote: Resp. bronchiles. Do u guys ever sleep???????????????
yes I was sleeping till 3pm..
total 9hrs of sleep.. 
Edited by kity on 09/10/07 - 02:11 PM
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| kity
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hope4dabest wrote: i knpw it sounds dumb....but whats medium bronchi????
its medium sized bronchi.. I just read it.. it said there are so many resp bronchiloes &its not the site of max airway resistance....
I am also wondering why??
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| LilInjnDatCould
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lilinjndatcould wrote:
here is my question: There are 3 main types of sphingolipids. What are they and please give an example of a genetic defect that results in accumulation of each type of sphingolipid.
Also can you all tell me the difference between a sphingolipid and a triglyceride?
i guess y'all didn't like my question. maybe not clinically relevant enough.. well, here is the answer nonetheless...
The difference between triglyceride and sphingolipids is the backbone is a serine instead of a glyecerol molecule.
Now the three major classes of sphingolipids differ by what is attached at the 3rd position of the Ceramide molecule (remember a Ceramide is a Serine backbone with 2 Fatty acids).
1)A ceramide with a simple sugar @ 3rd position (like a glucose or galactose) makes it a cerebroside. Deficiency/absence of glucocerebrosidase results in glucocerebroside in Gaucher's Dz! - rem: "crumpled or wrinkled paper" inclusions in lysosomes of their macrophages.
2)A ceramide with a complex sugar like @ 3rd postition (i.e an amino sugar) makes i a ganglioside. Deficiency/absence of HexosAMINIdase A results in Tay-Sachs dz with accumulation of Ganglioside GM2 - rem: "cherry red spot" of the macula in the retina. Also, Psychomotor retardation due to the the accumulation in the cns and thereby irreversibly neurological damage. Die b4 5 yrs age.
3)In the last class a ceramide complexes with a phopho-choline giving a sphingomyelin. Accumulation of sphingomyelins due to a deficiency of sphingomyelinase will result in Neimann-pick dz. These pts exhibit a mixture of both of the above Sxs. They also have Cherry red spot in macula, but they have a destinct "zebra body inclusions" on EM.
I guess it's a bad question, my bad.
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| hope4dabest
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nope the q is gr8...i didnt know the answer in such detail.. i had just done rattafication of the lysosomal disorders..
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| kity
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LilInjnDatCould wrote:
i guess y'all didn't like my question. maybe not clinically relevant enough.. well, here is the answer nonetheless...
The difference between triglyceride and sphingolipids is the backbone is a serine instead of a glyecerol molecule.
Now the three major classes of sphingolipids differ by what is attached at the 3rd position of the Ceramide molecule (remember a Ceramide is a Serine backbone with 2 Fatty acids).
1)A ceramide with a simple sugar @ 3rd position (like a glucose or galactose) makes it a cerebroside. Deficiency/absence of glucocerebrosidase results in glucocerebroside in Gaucher's Dz! - rem: "crumpled or wrinkled paper" inclusions in lysosomes of their macrophages.
2)A ceramide with a complex sugar like @ 3rd postition (i.e an amino sugar) makes i a ganglioside. Deficiency/absence of HexosAMINIdase A results in Tay-Sachs dz with accumulation of Ganglioside GM2 - rem: "cherry red spot" of the macula in the retina. Also, Psychomotor retardation due to the the accumulation in the cns and thereby irreversibly neurological damage. Die b4 5 yrs age.
3)In the last class a ceramide complexes with a phopho-choline giving a sphingomyelin. Accumulation of sphingomyelins due to a deficiency of sphingomyelinase will result in Neimann-pick dz. These pts exhibit a mixture of both of the above Sxs. They also have Cherry red spot in macula, but they have a destinct "zebra body inclusions" on EM.
I guess it's a bad question, my bad.
not bad Qn at all.. & very high yield.. but I had no clue as I am always confused in this n leave it for last min cramming..
Thanks fro posting the answe..
keep theQn coming
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