mytime Go Marching in!

Topics: 40 Posts: 3,177
| | 08/22/07 - 06:27 PM  
 
   
 
|   #51 |
  
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| edie I can, and I will.

Topics: 26 Posts: 1,202
| | 08/22/07 - 07:37 PM  
 
   
 
|   #52 |
Thanks for all the input, docs, very helpful Let's keep going! I need to go off, but will leave these, kinda easy, maybe. Anyone can answer. Pls leave a q if you want to 1. Which lung cancer is not associated with smoking? 2. Which lung cancer is associated with Lambert-Eaton syndrome? 3. Which of the pneumoconioses are associated with lung cancer? 4. What hormone(s) elevated in a lung cancer paraneoplastic syndrome?
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| new_n_lost Forum Hero

Topics: 663 Posts: 6,107
| | 08/22/07 - 07:53 PM  
 
   
 
|   #53 |
edie wrote:Thanks for all the input, docs, very helpful  Let's keep going! I need to go off, but will leave these, kinda easy, maybe. Anyone can answer. Pls leave a q if you want to 1. Which lung cancer is not associated with smoking? 2. Which lung cancer is associated with Lambert-Eaton syndrome? 3. Which of the pneumoconioses are associated with lung cancer? 4. What hormone(s) elevated in a lung cancer paraneoplastic syndrome? 1. Which lung cancer is not associated with smoking? Adenocarcinoma and Bronchial Carcinoid 2. Which lung cancer is associated with Lambert-Eaton syndrome? Small Cell Lung Ca 3. Which of the pneumoconioses are associated with lung cancer? All Except Silicosis 4. What hormone(s) elevated in a lung cancer paraneoplastic syndrome? ADH and /or ACTH
___________________ FORUM RULES-- Those who believe in telekinesis, raise my hand. I get enough exercise just by pushing my luck --P4U World.." The pure and simple truth is rarely pure and never simple."
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| mytime Go Marching in!

Topics: 40 Posts: 3,177
| | 08/22/07 - 09:27 PM  
 
   
 
|   #54 |
1.Adeno can be these days NNL......so stick to carcinoid!!!!!!!!!!! Remember the filters and all! 2. 3. Silicosis possibility of Tb. 4.And PTHr in Squamous cell Ca.
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| keepgoing Forum Fanatic

Topics: 72 Posts: 1,995
| | 08/22/07 - 09:48 PM  
 
   
 
|   #55 |
wow great show,i missed it all,but very well put up questions and answers..overall a great discussion love this kindda stuff!!thanks evryone!!!
___________________ "Deh Shiva Var Mohe Ahey ,Shubh Karman Te Kabhun Na Tarun ,Na Darun Arson Jab Jaye Laroon, Nischey Kar Apni Jeet Karoon"
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| paheli It'sAllAboutGoodKarma

Topics: 192 Posts: 2,575
| | 08/23/07 - 08:33 AM  
 
   
 
|   #56 |
wow, this is good work, guys!! i hope to know enough to join soon. i actually need a pencil and paper to jot down some stuff!!! absolutely love it!! thanks, everyone!
___________________ Prepare as if you're the worst, Perform as if you're the best! As you dream, so you manifest. So, DREAM BIG!! When you face hardship, remember, God never gives you more than you can handle. Keep your face to the sunshine and you cannot see the shadows.
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| mikky Forum Elite

Topics: 17 Posts: 418
| | 08/24/07 - 03:42 AM  
 
   
 
|   #57 |
Which tumor suppressor gene is is considered the "guardian of the genome" and why? What multiple-tumor syndrome is associated with this gene? my guess p-53, li--fraumnni syndrome
___________________ it is our choices that show what we truly are, far more than our abilities!!!!
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| po Forum Elite
Topics: 39 Posts: 356
| | 08/24/07 - 09:15 AM  
 
   
 
|   #58 |
anmelie wrote: Integration of HBV DNA into the host genome occurs at early steps of clonal tumour expansion, and it has been shown to induce direct insertional mutagenesis of diverse cancer-related genes in a number of cases.
know the diff betn HBV and HCV=[I got q bank Q wrong..so.] The precise mechanism by which HCV causes HCC is not known. Unlike the hepatitis B virus (HBV), HCV is not a DNA virus and does not become integrated within the genome of hepatocytes. It is more likely that HCC occurs against a background of inflammation and regeneration, associated with liver injury due to chronic hepatitis. Most, but not all, cases of HCV-related HCC occur in the presence of cirrhosis, suggesting that it is the underlying liver disease per se that is the risk factor for HCC rather than HCV infection. (4) The prevailing hypothesis has been that some cirrhotic nodules which grow larger than others (referred to as adenomatous hyperplasia) were the precursor for HCC. Recently, however, it has been suggested that foci of transformed hepatocytes may arise in between cirrhotic nodules and grow to become adenomatous hyperplasia and, eventually, HCC. http://hepatitis-c.de/carcinom.htm
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