drduck wrote:
the Q was simple.......it is TR...
TR has incompetence of tricuspid valve.....it means rt. ventricle not able to empty it self completely, due to incomplete closure of the tricuspid valve.
this leads to increase rt. ventricular end diastolic and end systolic volume, which occurs as a result of back log of previous cardiac cycle and gets added to the subsequent.
this incompitence increases progressively and so the end syst and diastolic volume....so the same number of rt. venricular muscle fibres have to work double or even more....to push the extra amount of load.
if u have understood this much no problem further...
extra work to same number of fibres....more chance of getting hypertrophied.....that is rt. ventricular hypertrophy.....still more work.....no hypertrophy possible..........FAILURE....both of which are evident on radiography...
now, rt. ventr. has failed so even less blood is pushed in the pulmonary circuit.....regurges back into the rt. atrium....cannot hypertrophy so goes in failure........this leads to falling back of pressure on to IVC.....so LIVER DISTENSION....
pressure on SVC.........so EXT JUG VEIN DISTENTION...
elevation of rt. atrial pressure..........DIST OF AZYGOUS VEIN>>>>>>>>PLEURAL EFFUSION.

and yes there wont be any left atrial pressure elevation or failure.......
bcos less blood reaching pulmonary circuit............less blood to left atrium........rather there will be left atrial and ventricular atrophy....partially.......

left sisded failure may lead to rt. sided failure bur rt. sided failure never leads to left sided failure.....

hope that helps.......