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Kaplan Qbank USMLE



Author9 Posts
  #1


lets see whos giving it a shot smiling face

18 y.o female c/o fatigue, acnea , hirsutism. mild hypertensive, hypo-k emia, plasma renin decr. serum corticotropin incr, urinary free cortisol incr. whas the deficiency?


Edited by Luckyall on 05/25/07 - 12:50 AM

  #2

need a clarification luckyall: are u talking about urinary free cortisol or its metabolites (17OH steroids)??

___________________
Everything turns out ok in the end. If it's not ok, then it's not the end.

  #3

urinary free cortisol...

  #4

if urinary free cortisol is increased, then the pathology must lie at the glucocorticoid receptor

___________________
Everything turns out ok in the end. If it's not ok, then it's not the end.

  #5

silver wrote:
if urinary free cortisol is increased, then the pathology must lie at the glucocorticoid receptor

nodnodnod Agree

___________________
FORUM RULES-- Those who believe in telekinesis, raise my hand. I get enough exercise just by pushing my luck --P4U World.." The pure and simple truth is rarely pure and never simple."

  #6

Answer plz ??smiling face

___________________
FORUM RULES-- Those who believe in telekinesis, raise my hand. I get enough exercise just by pushing my luck --P4U World.." The pure and simple truth is rarely pure and never simple."

  #7

what happened to this scenario? raised eyebrowcoolsmiling face

___________________
Everything turns out ok in the end. If it's not ok, then it's not the end.

  #8

Good job, guys! wink
Full Glucocorticoids( GC) resistance is lethal, but partial GC resistance causes a compensatory incr ACTH ---> incr Cortisol production---> maintaining normal Cortisol effects.
Incr. ACTH--->Incr. Adrenal Androgens prod---> acnea& hirsutism
Incr ACTH----->incr. steroid precursors---> incr MineraloCortic. effects ---> HTN & hypoK--emia---> decr Renin
Good job!

  #9

Thanks for the scenarios Lucky. Keep up the good job everyone!







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