Ancylostoma Forum Guru
Topics: 42 Posts: 637
| | 01/31/07 - 07:43 PM  
 
   
 
|   #1 |
When a 25 year onld patient was given oral glucose his insulin rose more then parental glucose. What is main factor responsible for this difference?
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| Clozapine Forum Elite

Topics: 15 Posts: 164
| | 01/31/07 - 07:55 PM  
 
   
 
|   #2 |
due to effect mediated by gut hormones ?
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| new_n_lost Forum Hero

Topics: 663 Posts: 6,107
| | 01/31/07 - 09:18 PM  
 
   
 
|   #3 |
Yes i wud say the same thru glucagon-like peptide-1 a gut hormone
___________________ FORUM RULES-- Those who believe in telekinesis, raise my hand. I get enough exercise just by pushing my luck --P4U World.." The pure and simple truth is rarely pure and never simple."
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| me007 Forum Guru
Topics: 72 Posts: 799
| | 01/31/07 - 09:55 PM  
 
   
 
|   #4 |
GIP = glucose-dependent insulinotropic peptide only GI hormone released in response to fat, protein and carbs
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| Ancylostoma Forum Guru
Topics: 42 Posts: 637
| | 01/31/07 - 10:17 PM  
 
   
 
|   #5 |
Thats right, while the there may be other hormones that are stimulate insulin secretion, the main one, is Gastric inhibitory peptide ( aka glucose-dependent insulinotropic peptide ). as a correlation, what effect would the removal of the duodenum have on gastric motility and why?
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| me007 Forum Guru
Topics: 72 Posts: 799
| | 01/31/07 - 10:48 PM  
 
   
 
|   #6 |
postprandial diarrhea. why - bile salts ?
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| Ancylostoma Forum Guru
Topics: 42 Posts: 637
| | 01/31/07 - 11:01 PM  
 
   
 
|   #7 |
There would be a large increase in gastric motilty, because the lack of the I cells of the duodenum. the I cells produce cck, and under physiologic conditions, are the only inhibitor of gastric motility. If you are searching for this fact in kaplan notes you won't find this exact line. Its something that the physiology q banks clarifys.
Edited by Ancylostoma on 01/31/07 - 11:10 PM
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| Ancylostoma Forum Guru
Topics: 42 Posts: 637
| | 01/31/07 - 11:43 PM  
 
   
 
|   #8 |
Making my point that it might actually be worth doing !!
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