AAAAA
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A
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| AAAAA
Forum Fanatic
Topics: 156
Posts: 1,991
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I am somewhat biased against beta adrengeric blocking agents. Once you put a patient on beta-blocker, you may have to put the patient for life of these kind of medication.
They get reflex tachycardia if the medication is stopped.
Also high dose of beta-blockers may worsen depression in those who are depressed, decrease reaction time and actually may be used in anxiety.
The use atenolol: Atenolol is a beta-adrenergic blocking agent. Atenolol blocks the action of the sympathetic nervous system, a portion of the involuntary nervous system. The sympathetic nervous system stimulates the pace of the heart beat. By blocking the action of these nerves, atenolol reduces the heart rate and is useful in treating abnormally rapid heart rhythms. Atenolol also reduces the force of heart muscle contraction and lowers blood pressure. By reducing the heart rate and the force of muscle contraction, atenolol reduces heart muscle oxygen demand. Since angina occurs when oxygen demand of the heart exceeds supply, atenolol is helpful in treating angina.
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| sunny2
Forum Elite
Topics: 51
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s2006 the answer is - A , and the logic is :
B-blockers decrease heart rate, blood pressure and contractality so decreases myocardial O2 requirement.
also remember that B-blockers - Increase preload, EF, CO and decreases mortality in MI and CHF ...and a little clinical pearl is that it is used in MVP and HOCM
hope that helps 
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| s2006
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Topics: 17
Posts: 39
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WONDERFUL EXPLANATION, THANX AAAAA&SUNNY2
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