Leopard
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jamdoc wrote:
Pulse pressure is proportional to the amount of blood entering the aorta during systole and inversely proportional to aortic compliance. Pulse pressure increases with hypertension because hypertension causes aortic compliance to decrease. Whether the hypertension is a result of an increased cardiac output or an increased peripheral resistance, the higher arterial pressure is caused by an increase in arterial'blood volume.
Jamdoc
This is strange information.
You have a normal person ,you increase his MAP with some drug ,so you mean its compliance is reduced immediatlely ??? How ?? Would you please explain??
Yes Long standing cases may lead to atherosclerosis ia a complication which may reduce compliance but not immediatley .What is the source of this question.??? Thanks in advance
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| jamdoc
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http://circ.ahajournals.org/cgi/reprint/107/22/28...
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| jamdoc
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http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?ito...
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| Leopard
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These are seven pages.
Can you please copy paste the relevant material (sentences) which says that HTN decreases compliance ??
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| jamdoc
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in the above articles, its mainly emphasised abt isolated systolic htn but i guess if we have to choose amogst the best,i would go for hypertension
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| Leopard
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jamdoc wrote:
in the above articles, its mainly emphasised abt isolated systolic htn but i guess if we have to choose amogst the best,i would go for hypertension
The question is How ???
Three factors control PP
- Central
- Stroke volume -- direct proprotionality
- compliance -- inverse proportionaliy
- Peripheral
- TPR by controlling DBP
In fact ,if you look at pressure volume loop of the heart ,stroke volume is subtantially decreased when we increase After load (MAP )
But Your claim is HTN decreases Compliance .My Question is How ?? Logic or reference ?
Thanking in anticipation .
Best of luck
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| ssrpk
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whtz the source of this question btw? the answer is just too controversil as well as versatile in this sense tht all the explanations wud make sense, like HTN if assicated with long term atherosclerosis will make arteries stifer, but then lots and lots can happen, on one hand low compliance mean mean high systolic and low diastolic, whle on the other hand increase afterload means low peak pressure which will reduce systolic pressure!
all in all, these kind of questions are unfair! where u don't hv any definite objective conclusion or different possiblities!
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| jamdoc
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hi guys i appoligize for posting such question,but i wanted an explanation,thought i could get some feedback from u guys.anyway just copying a link from a website.btw this q is from score95.they have some weird questions.
Section of Hypertension and Clinical Pharmacology, University of Connecticut School of Medicine, 263 Farmington Avenue, Farmington, CT 06030-3940, USA. wwhite@nso1.uchc.edu
Elevation of systolic blood pressure (BP) has been recognized as an independent risk factor that far exceeds the risk associated with an elevated diastolic BP in older patients with hypertension. Isolated systolic hypertension (ISH) is a disorder typically defined when the systolic BP is greater than 140 mm Hg but with diastolic BP below 90 mm Hg. Pulse pressure (the difference between systolic and diastolic pressure) has recently become an active area of discussion in the literature as an independent factor of cardiovascular risk. An increased pulse pressure nearly always indicates reduced vascular compliance of large arteries and, by definition, is always increased in patients with isolated systolic hypertension. Although the evidence that a widened pulse pressure is an independent marker of cardiovascular risk is quite well established, therapeutic morbidity and mortality studies in ISH have focused on reductions in systolic pressure. At the present time, outcomes data have not been well established for reductions in pulse pressure in older patients with hypertension.
PMID: 12379164 [PubMed - indexed for MEDLINE]
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