smal wrote:
thanks musuq
can u plz tell if u got the pic from which website.i've been searching for something in animation that would improve my understanding of ANS.
smal wrote:
thanks musuqcan u plz tell if u got the pic from which website.i've been searching for something in animation that would improve my understanding of ANS.
Here's how I learnt about it...
Sympathetic nervous system:
<>--------< (N) <>--------< (A) [muscle]
Parasympathetic nervous system:
<>---------< (N) <>-----------< (M) [muslce]
N = nicotinic acetylcholine receptors
A = adrenoreceptors (NB. Noradrenaline is the neurotransmitter here - not acetylcholine)
M = muscarinic aceylcholine recepteors
Btw, tubocurarine doesn't have any effect on the autonomic nervous system!...
In this experiment, we are talking about skeletal muscle being innervated by somatic nerves (not autonomic nerves)
<>-----------------------< (N) [muscle]
Tubocurarine competes for ACh for the nicotinic acetylcholine receptor...
Neostigme is an anti-cholinesterase... acetyl-cholinesterase breaks down ACh on the nicotinic acetylcholine receptor... Therefore, if you inhibit this cholinesterase with neostigme, you are going to cause ACh to bind longer to the nAChR... This is why the resoponce in the muscle increases...
Makes sense?
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