drfax wrote:
It is Amiodarone, I'm sure look up at Kazung's pharmacology review 7th. P.128
BUT Y ?????
- The acquired conditions that predispose one to torsade either decrease the outward potassium current or interfere with the inward sodium and calcium currents, or fluxes.
- The electrolyte disturbances that have been reported to precipitate torsade include hypokalemia and hypomagnesemia. Close observation is required in predisposed patients, such as those with cirrhosis or hypothyroidism.
- Hypokalemia and hypomagnesemia, in turn, cause a delay in phase III (ie, reprolongation) and form the substrate for emergence of the dysrhythmia.
- Antiarrhythmic drugs reported to be etiologic include class IA agents (eg, quinidine, procainamide, disopyramide), class IC agents (eg, encainide, flecainide), and class III agents (eg, sotalol, amiodarone).
http://www.emedicine.com/emerg/topic596.htm
Best of luck
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| docswapna
Forum Senior
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so its not amiodarone. wats the ans drkaur?
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| drpkaur
Forum Guru
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it's F...GUYS...
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