Prep for USMLEPrep for USMLE Forum
   Forum    Step 1  Step 2 CK Step 2 CS Step 3  Match  IMGs Resources Search






Previous Topic | Next Topic  visual 




 
Kaplan Qbank USMLE



Author9 Posts
  #1

After nearly drowning, an 70-year-old woman suffers from severe hypoxia with resulting severe brain edema. This edema leads to raised intracranial pressure. When the pressure exceeds 200 mmHg, the medial temporal lobe begins to be compressed against the tentorium cerebelli. What is typically the first sign of such compression?

A. Anisocoria contralateral to compression

B. Anisocoria ipsilateral to compression

C. Both pupils 5–6 mm and fixed

D. External oculomotor ophthalmoplegia

E. This condition has no effect on the eyes


  #2

B raised eyebrow

  #3

A and B
Anisocoria means asymmetric pupil size...not whcih one is small or large...
so both A and B!!!

  #4

Dr.papez without taking choices into consideration, can u plz explain what actually happens in this situation (constriction,dilation etc)

thanku.


  #5

I agree with Dr Papez in this regard.Can anybody else throw more light on this topic

___________________
FA is just a good revision book.It is not a "real" learning tool.

  #6

B
Uncal herniation
In uncal or transtentorial herniation, the innermost part of the temporal lobe, the uncus, can be squeezed so much that it goes by the tentorium and puts pressure on the brainstem (Shepherd, 2004). The tentorium is a structure within the skull formed by the meningeal layer the dura mater. Tissue may be stripped from the cerebral cortex in a process called decortication (McCaffrey, 2001). The uncus can squeeze the third cranial nerve, which controls parasympathetic input to the eye on the side of the affected nerve. This interrupts the parasympathetic neural transmission, causing the pupil of the affected eye to dilate and fail to constrict in response to light as it should, so a dilated unresponsive pupil is an important sign of increased intracranial pressure (Cornell, 1998). Cranial arteries may be compressed during the herniation (Orlando Regional Healthcare, 2004). This type of herniation can also damage the brain stem, causing lethargy, slow heart rate, respiratory abnormalities, and pupil dilation (McCaffrey, 2001). Uncal herniation may advance to central herniation


  #7

Thanx for the explanation vanshita

___________________
Aim High

  #8

vanshita wrote:
B
Uncal herniation
In uncal or transtentorial herniation, the innermost part of the temporal lobe, the uncus, can be squeezed so much that it goes by the tentorium and puts pressure on the brainstem (Shepherd, 2004). The tentorium is a structure within the skull formed by the meningeal layer the dura mater. Tissue may be stripped from the cerebral cortex in a process called decortication (McCaffrey, 2001). The uncus can squeeze the third cranial nerve, which controls parasympathetic input to the eye on the side of the affected nerve. This interrupts the parasympathetic neural transmission, causing the pupil of the affected eye to dilate and fail to constrict in response to light as it should, so a dilated unresponsive pupil is an important sign of increased intracranial pressure (Cornell, 1998). Cranial arteries may be compressed during the herniation (Orlando Regional Healthcare, 2004). This type of herniation can also damage the brain stem, causing lethargy, slow heart rate, respiratory abnormalities, and pupil dilation (McCaffrey, 2001). Uncal herniation may advance to central herniation



Your explanation is very good.But now the question is whether Anisocoria can be labelled as Ipsilateral or contralateral.Please throw some light on this disputed point.Please do help othewise my brain is melting and neurons are getting lost.

Thanks in advance.


___________________
FA is just a good revision book.It is not a "real" learning tool.

  #9

in this pt compression of prasypathatic fibres of occulomotar nerve cause dillation of ipsilateral pupil
so answer is B anisocoria ipsilateral side







You don't have permission to post.




Login or Register to post messages in this topic





















Contact | Leaders | Disclaimer | Privacy

Copyright @ Prep for USMLE. All rights reserved.