drpkaur
Forum Guru
Topics: 196
Posts: 810
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1. Would blockade of muscarininc receptors in the bladder be useful in treating urinary retention?
2. Why is there a drop in systolic, mean, and diastolic bp with infusion of isoproterenol?
3. Why does NE result in bradycardia?
4. Which drug increases Sys BP w/o affecting Pulse Pressure?
5. Which antimuscarinic agents are used in producing mydriasis and cycloplegia?
6. What type of neurological blockade would hexamethonium create?
7. What is the difference in receptor affinity of epinephrine at low doses? High doses?
8. What is the clinical utility of clonidine?
9. What effect would atropine have on a patient with peptic ulcer disease?
10. What are the phases of succinylcholine neuromuscular blockade?
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| mallik
Forum Newbie
Topics: 0
Posts: 4
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9 Atropine decrease gastric secretion and give symptomatic relief in peptic ulcer disease
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| suv
Forum Elite
Topics: 43
Posts: 233
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2.NE causes increase in BP so there is reflex bradycardia
5.Atropine
6.ganglion blockade competitive antagonists at NN receptors
7beta predominance at low doses and alpha predominance at high doses
8.alpha 2 agonist decrease vasomotor outflow
used in mild to moderate HTN
9. atropine inhibits gastric acid secretion at high doses and is used in the mangement of peptic ulcers
10. Succinyl choline is a depolarizing neuromuscular blocker.
Phase 1- Prolonged Depolorization - Block potentiated by cholinestrease inhibitors.
Phase -2 - Repolarized but blocked - Antidote Neostigmine.
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