study_ing Forum Fanatic

Topics: 180 Posts: 2,511
| | 07/21/06 - 11:28 AM  
 
   
 
|   #26 |
for acute management of av nodal yes (3)
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| pallav Forum Elite

Topics: 56 Posts: 180
| | 07/21/06 - 10:17 PM  
 
   
 
|   #27 |
boy you are intelligent fellas i dont know whats gonna happen to people like me luv pallav
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| khorshid Forum Guru
Topics: 32 Posts: 591
| | 07/22/06 - 04:33 PM  
 
   
 
|   #28 |
Here are some more: -which one should not be given in myasthenia?-------------------Quinidine( opposes ACh inh) -which antiarrhythmatic may cause SLE like syn?------Procainamide (pro drug metabolized by N-acetyltransferase, therefore can't be used IV) - class IB has more affinity for partly depolarized tissues=ischemic tissues, I guess that's why they are used post MI (like lidocaine) -acute SVT-----------------IV esmolol (class II), digoxin is also used -Amiodarone---------------check PFT,LFT,TFT -Amiodarone----------------corneal deposition and smurf skin(blue pigmentation) -Avoid CCAs in V tach-------------may turn into VF -which drug antogonizes adenosine?--------------------theophylline.
Edited by khorshid on 07/22/06 - 05:00 PM
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| young_doc Forum Guru

Topics: 55 Posts: 732
| | 07/25/06 - 03:27 PM  
 
   
 
|   #29 |
Antiarrhythmics never made sense to me until i watched the Pass videos. If any of you have them just sitting on your computer, at the very least watch the Antiarrhythmic's part of it! You'll be glad you did. WPW: ANYTHING that delays A-V conduction (hence b-block, dig, Ca-block, M2-agonist, you name it!!!!!!!!!!). And the 'abberrant pathway' everyone's talking about, don't forget what it's called!!! Bundle of Kent. It's the reason why we see the Delta wave (pathognomic for WPW). good thread though!!
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| Chrales Forum Newbie
Topics: 2 Posts: 30
| | 07/27/06 - 08:06 AM  
 
   
 
|   #30 |
treatment WPW: K+ channel-blockers (class III) -->increasing refractory period
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| study_ing Forum Fanatic

Topics: 180 Posts: 2,511
| | 08/02/06 - 01:02 PM  
 
   
 
|   #31 |
what kinds of arrythmias are seen with digoxin? With toxicity, practically any arrhythmia can be seen, although certain arrhythmias are highly suggestive, for example, the presence of both increased irritability and AV nodal block (such as paroxysmal atrial tachycardia with a 2:1 AV nodal block).
Edited by study_ing on 08/02/06 - 01:08 PM
___________________ If you yourself are at peace, then there is at least some peace in the world.
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| study_ing Forum Fanatic

Topics: 180 Posts: 2,511
| | 08/09/06 - 04:55 PM  
 
   
 
|   #32 |
how do we manage digoxin toxicity? how does k affect it? the Digitalis toxicity is treated by : 1-withdrawal of digitalis 2-give Digibind Fab if available 3- [K+] : msyamp : digitalis binds to the K+-part of the Na-K-ATPase. so, K+ can compete with Digitalis for the binding site and keep the pump runnig. 4- groupe II/b antiarrythmics : Lidocain and phenytoin
___________________ If you yourself are at peace, then there is at least some peace in the world.
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| suv Forum Elite
Topics: 43 Posts: 233
| | 08/14/06 - 12:37 PM  
 
   
 
|   #33 |
drugs causing torsades? ECG changes in torsades? treatment of torsades de pointes?
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| lilla Forum Senior
Topics: 26 Posts: 153
| | 09/11/06 - 03:35 PM  
 
   
 
|   #34 |
great job!!! drugs causing torsades: classIA: quinidine classIII: bretylium sotalol ibutilide amiodarone classIV:bepridil phenothiazines cisapride
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| suv Forum Elite
Topics: 43 Posts: 233
| | 09/13/06 - 07:23 AM  
 
   
 
|   #35 |
ecg changes in torsades prolonged QT interval treatment iv magnesium, isoproterenol and electrical pacing
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