Ms.Apara Forum Senior
Topics: 31 Posts: 128
| | 06/01/06 - 11:47 AM  
 
|   #2 |
A
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| star1 Forum Guru

Topics: 93 Posts: 827
| | 06/01/06 - 11:49 AM  
 
|   #3 |
A
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| nadiabarati
| | 06/01/06 - 11:56 AM  
 
|   #4 |
upon deoxidation HbS can not produce tetramers and rather forms long polymers. So I think A and C are right! What is wrong with my thought?!!
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| Slacker Forum Elite
Topics: 29 Posts: 141
| | 06/01/06 - 02:07 PM  
 
|   #5 |
Why can't it be D. The HbS are instable and that causes spontaneous sickling of the RBCs. Also low PaO2 can cause sickling in a person with Sickle cel trait.
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| Cedrick Esculapio

Topics: 324 Posts: 2,058
| | 06/01/06 - 02:45 PM  
 
|   #6 |
it bends and deforms the cell, A
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| xwxf Forum Senior

Topics: 16 Posts: 71
| | 06/01/06 - 07:02 PM  
 
|   #7 |
HBS can polymerise upon low O2 tension or hypotonic stress. The polymerized HBS deforms RBC.
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| DrVirgo Forum Hero

Topics: 1096 Posts: 3,515
| | 06/09/06 - 09:04 PM  
 
|   #8 |
Where can we look this up? I think its either C or D. Factors that induce sickling are: -HbS in RbC>60% -Reduced O2 tension -High altitude -Low O2 tension in the renal medulla etc.... But what about the structure of the Hb?
___________________ Our greatest glory is not in never falling, but in rising every time we fall.
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