robin082006
Forum Hero
Topics: 471
Posts: 5,123
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D
___________________
The Key to Succeed is Patience.
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| r_albayunen
Forum Senior
Topics: 0
Posts: 218
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d
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| star1
Forum Guru
Topics: 93
Posts: 827
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D 
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| nadiabarati
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secondary to what?!!!
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| star1
Forum Guru
Topics: 93
Posts: 827
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ohhhhhh
In liver disease protein are not produced and so colloidal osmotic pressure decrease and this lead to Edema . As fluid is leaving the vascular compartment so Vascular volume decreases and this will stimulate Renin- Angiotension Aldosterone system to increase Na reabsorbtion and ultimately increase the severity of edema. We give Na to supress the Aldosterone secretion .
Correct me if wrong.
thanks
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| nadiabarati
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But why we don't say that in liver disease we have edema and this edema suppress renin secretion,because aldosterone not only deals with intravascular volume but also with interstitial volume. So why increase volume of interstitium doesn't supress aldosterone secretion??!
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| frontal
Forum Guru
Topics: 53
Posts: 421
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ladybug, do you have the answer? I'll guess B.
'hepatic liver' disease. lol. You could've omitted 'hepatic'.
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| ladybug
Forum Senior
Topics: 20
Posts: 57
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Secondary hyperaldosteronism is generally related to hypertension (high blood pressure) and swelling disorders, such as cardiac failure, CIRRHOSIS of the liver, and nephrotic (kidney) syndrome. In these disorders, various mechanisms from the individual disease process cause the level of the hormone to be elevated.
The excess aldosterone secreted in these conditions increases sodium reabsorption and potassium loss by the kidneys and results in electrolyte imbalances.
Answer is D.
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