reet
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cGMP role IN ROD CELLS in vision ????can anyone explain plz
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| reet
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ok got it myself
in response to light-- pathway activates cGMP phosphodiesterase which lower cGMP and hyperpolarise the CELL MEMB OF outer rod segment.
in dark--- high cGMP-- DEPOLARISE memb
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| reet
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how the LIGHT activated signal is going to brain???? plz help me
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| druas
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hey reet r u talking to yourself
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| reet
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may be the question i asked ,,doesnt make sense..
i asked about the substance whic h is released during HYPERPOLARISATION... --and sending signal to brain... is GLUTAMATE i.e it relieving the inhibition of optic nerve bipolar cells and intiating singnal to brain
Edited by reet on 02/07/06 - 07:36 PM
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| reet
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Well,,thats wht i couldnt understand first..
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| Geroo
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is GLUTAMATE inhibitory or exitatory?
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| reet
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rod/cones RELEASE glutamate is either excitates or inhibits the BIPOLAR cells they synapse on
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| mildus
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there are several opinions (just compare Guyton and Ganong Med. physiology)
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| mildus
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One theory:
rods/cones release inhibitory neurotransmiter, but when they hyperpolarize, releasing of inhibitory neurotransmiter stops, so you have stimulation of bipolar cells
Other theory:
when rodes/cones hyperpolarize, they start releasing glutamate which stimulates bipolar cells
I'm more familiar with the first one
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| mildus
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I don't know what is the true
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