drk1980
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Beta2-agonists are involved in transcellular shift of potassium. Shift in or shift out?
One of the negative impacts of hypokalemia is diabetes insipidus. Nephrogenic or Central?
Cardiac Arrest is in DIASTOLE when associated with hyperkalemia. In systole when a/s wth wht?
One of the methods of management in some cases of potassium derangements is Calcium Gluconate. Does it increase or decrease levels of potassium?
thanks
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| skyhigh
Forum Guru
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beta 2 agonists allow transcellular shift of potassium into cells
negative impact of hypokalemia is Nephrogenic diabetes insipidus
cardiac arrest in systole is associated with increased levels of calcium
Calcium gluconate causes shift of potassium into cells, so i think it decreases potassium levels
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| skyhigh
Forum Guru
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plz correct me if wrong
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| drk1980
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excellent!
beta agonists are a cause of hypokalemia and beta blockers, just the opposite.
hypokalemia has an effect of vacuolar nephropathy, making the collecting ducts refractory to ADH.
cardiac arrest in systole, ATP is depleted leaving calcium in charge without any use.
the last one ws a bit of a googly. In hyperkalemia, when you find ECG changes, to prevent cardiotoxicity you shd administer calcium gluconate/chloride to stabilize the cardiac membranes. Has no effect as such on potassium levels.
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| robin082006
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good query and good explanation
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