chemamr
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Tick paralysis occurs when an engorged and gravid (egg-laden) female tick produces a neurotoxin in its salivary glands and transmits it to its host during feeding. Experiments have indicated that the greatest amount of toxin is produced between the fifth and seventh day of attachment (often initiating or increasing the severity of symptoms), although the timing may vary depending on the species of tick.
Unlike Lyme disease, ehrlichiosis, and babesiosis, which are caused by the systemic proliferation and expansion of parasites in their hosts long after the offending tick is gone, tick paralysis is chemically induced by the tick and can therefore continue only in its presence. Once the tick is removed, symptoms usually diminish rapidly and that is the treatment. However, in some cases, profound paralysis can develop and even become fatal before anyone becomes aware of a tick's presence.
In the United States, this disease is associated with Dermacentor andersoni (Rocky Mountain wood tick), D. variabilis (American dog tick), Amblyomma americanum (Lone Star tick), A. maculatum, Ixodes scapularis (black-legged tick), and I. pacificus (western black-legged tick).
Hope this helps.
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| LisaM
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Thank you, Chemamr. It sure helps!
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| alenka
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i had similar q on UW-it present just like gullian-barre -ascending paralysis.the only difference-no history of diarrhea at all.plus the tick paralysis develops faster.
just know one thing-they might not give you history of camping trip,like was with this q on UW-but you can't dismiss the suspect because of this.
plus the differential with botulism -in botulism there's a descending,not ascending paralysis.
by the way-i missed that question
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