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Kaplan Qbank USMLE



Author8 Posts
  #1

CONJUGATED VACCINE ALLOWS B CELL DIFFERENCIATION AND CONSEQUENTLY THE RESPONSE WILL HAVE IGG AND IGA OR IF NO CONJUGATION : STILL RESPONSE BUT ONLY IGM


  #2

GUY'S WIFE HAS ')">shockedSTD
: IF YOUR A GUY YOU TELL ON HER !!!!! IF YOUR A WOMAN
YOU TELL ON HER !!!!!! IF YOU WANT TO
AVOID GOING TO COURT AN
D RESPECT DR-PATIENT CONFIDENTIALITY YOU SHUT UP!!!!! OR TRY TO GET HER TO SAY TO HIM')">sticking out tongue .....AND HE MAY KNOW WHY.......')">mad


Edited by David Noc on 09/22/05 - 01:40 PM. Reason: SMILES ADDED AND SPELLING

  #3

There is considerable potential for iron toxicity because very little iron is excreted from the body. Thus, iron can accumulate in body tissues and organs when normal storage sites are full. For example, people with hemachromatosis are at risk of developing iron toxicity because of their high iron stores.

In children, death has occurred from ingesting 200 mg of iron . It is important to keep iron supplements tightly capped and away from children's reach. Any time excessive iron intake is suspected, immediately call your physician or Poison Control Center, or visit your local emergency room. Doses of iron prescribed for iron deficiency anemia in adults are associated with constipation, nausea, vomiting, and diarrhea, especially when the supplements are taken on an empty stomach .

In 2001, the Institute of Medicine of the National Academy of Sciences set a tolerable upper intake level (UL) for iron for healthy people . There may be times when a physician prescribes an intake higher than the upper limit, such as when individuals with iron deficiency anemia need higher doses to replenish their iron stores. Table 5 lists the ULs for healthy adults, children, and infants 7 to 12 months of age . FROM NIH



  #4

The ingestion of a large amount of iron overwhelms gastrointestinal regulatory mechanisms resulting in massive iron absorption. Following absorption, when serum iron levels exceed the amount of binding protein, transferrin (TIBC), severe toxicity may occur secondary to deposition of iron in soft tissues. The exact mechanism of toxicity at the cellular level is unknown but is likely due to direct cellular oxidative damage.FROM MD POISON CENTER

b. peroxidation of DNA

XX c. peroxidation of lipid....SEEMS THE MOST LOGICAL SINCE damage to DNA would mean the iron is transported into the cell and nucleus


  #5

#4- DKA is trigered by infection, stressful situation lead to inc. in stress hormones particularly cortisol levels which leads to inc. in gluconeogenesis and exceeseivly hgh glucose levels in the face of IDDM.

#3- agree with david, it's confidential!


___________________
life is guud

  #6

does anyone remember the picture of the hand having a few pus filled lesions..what was that?on nbme form 2

  #7

well the picture looks a lot like the one on the exam..so it must be a vacuolated squamous cell..doesnt look much like a blister to me..anyone else has an opinion to this

i think the nbme questions r same for all who take it except for the order of the questions..

do u remember the dumb question where they asked abt a lady who had an allergy to sunscreen containing methylparaben..any idea what the answer to that was?


Edited by sobia naeem on 09/23/05 - 02:00 AM

  #8

thanks for the answer...this question was on my form 2.







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