phuluong2k
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Premature labor with premature rupture of fetal membranes occurs at 20 weeks gestation in a 20-year-old woman. Prior to that time, the pregnancy had been proceeding normally. A stillbirth occurs two days later. Microscopic examination of the normal-sized placenta reveals numerous neutrophils in the amnion and chorion, but no villitis. These events are most likely to be mediated by the effects from release of which of the following substances?
A Immunoglobulin
B Prostaglandin
C Complement
D Fibrinogen
E Lymphokines
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| KEROCHI
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D fibrinogen
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| phuluong2k
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I think it is prostaglandin
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| dev
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are u sure its pgs?y not complement?
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| Kluver_Bucy
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I'm going with cytokines/lymphokines. This is what recruited the neutrophils there, and the rupture of the placental membrane.
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i think its complement and C5a acts in attracting the neutrophils....
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| merrk
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No vilitis...
Neutrophils are attracted by IL 8 and C5a and there would be inflammation.
I'd go for fibrinogen
abruptio placenta causes DIC from release of fibrinogen... (I think)
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| phuluong2k
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I have no clear answer for this q
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| yaku
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B Prostaglandin
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